It plays a critical role in detoxification, immune support, and cellular repair
Therefore, directing interventions toward iron metabolic pathways and modulating cellular iron homeostasis could present a promising avenue for breast cancer treatment, by utilizing strategies such as iron chelation and addressing iron overload
APCs present thyroid autoantigens to T cells via HLA class II molecules, providing the first activation signal through the T-cell receptor
These pathways can regulate metabolism and epigenetics and change the response of cancer cells to both targeted therapies and chemotherapies (Lee et al., 2020)
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In theory, this could include promoting the growth of existing tumours or disrupting normal endocrine function