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Characterization of the serotoninergic system in the C57BL/6 mouse skin

The toxic side of one-carbon metabolism and epigenetics

Adult restoration of Shank3 expression rescues selective autistic-like phenotypes

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These specialized enzymes transfer electrons from cytoplasmic NADPH to generate ROS that initiate lipid peroxidation

Evidence from multiple fields, including metabolism, cell biology, and clinical endocrinology, collectively suggests that within the uterine fibroid microenvironment, E 2 and progesterone may induce excessive ROS accumulation through three pathways: enzyme metabolic imbalance, mitochondrial dysfunction, and disruption of the antioxidant system