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The prevailing view is that Fer-1 resistance to ferroptosis is achieved by scavenging alkoxyl radicals and other rearranged products produced by ferrous ions from lipid hydroperoxides 18

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These data suggest that high glucose levels promoted the proliferation of activated hPSCs through the TGF signalling pathway, and GSH has the potential to attenuate the activation phenotype of hPSCs

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The protective mechanism of preconditioning involves the enhanced ROS-scavenging activity caused by the production of small amount of ROS by short-term ischemia (Morihira et al