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POLK overexpression contributes to cancer development by inactivating wild-type p53, and this was shown in lung cancer 47

The mechanism involves stimulating over 300 different genes associated with tissue repair and remodeling, fibroblast activation and collagen synthesis, epidermal basal stem cell viability, an anti-inflammatory effect, an antioxidant effect and stimulation of microvascular growth

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Meeting held at IARC, LYON, 714 june 1994

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In the upstream, chronic hypertension and diabetes (both common in cSVD) have been shown to impair cerebral blood flow and oxygenation and promote an increase in BBB permeability