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Morris: Good question

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On the other hand, after deacetylation of FOXO by SIRT1, FOXO can be degraded by ubiquitination, reducing the level of FOXO and inhibiting the ability of FOXO to induce cell death, thereby ultimately protecting cells from oxidative stress damage [47, 48]

It plays a crucial role in energy production by transporting fatty acids into your cells' mitochondria, which act much like engines within your cells

Therefore, ginsenosides mainly reduce the hepatotoxicity of APAP by inhibiting oxidative stress

Bisht S, Faiq M, Tolahunase M, Dada R