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Disorders of hemoglobin synthesis

While animal models have shown a favorable safety profile, and many individuals report positive experiences, the absence of extensive human clinical trials means that caution is advised

Notably, the study documents the negative regulatory function of TFR2 in TNBC ferroptosis, with TFR2 downregulation leading to inhibited proliferation and ferroptosis induction in TNBC (Yang et al., 2024a)

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The results showed that cells with suppressed GPX3 expression were more susceptible to cell death induced by ascorbate, indicating that GPX3 expression is crucial for scavenging excessive extracellular H2O2