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Structure-activity relationships in beta-defensin peptides

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Kim J, Yun JM, Kim MK, Kwon O, Cho B

Articles that did not involve or discuss about patients with pernicious anemia treated with oral vitamin B12 replacement were excluded

When cyp2e1 knockout mice that lack the ability to produce the CYP2E1 enzyme were challenged with acetaminophen, they were found to be considerably less sensitive to its hepatotoxic effects than wild-type animals, indicating that this CYP is the principal enzyme responsible for the metabolic conversion of the drug acetaminophen to its active hepatotoxic metabolite NAPQI (Figure 2)

That is a very different statement from they do nothing. It means: The mechanistic rationale is real