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However, when cells are exposed to excessive and/or prolonged ROS levels, which can cause uncontrollable damage, p53 activity is inhibited by ROS, leading to cell death pathways to protect adjacent undamaged cells (83,84)

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10.1111/j.1365-2133.2008.08717.x 37 ChaudharyS.DayalS

These observations support a model of APR-246 activity in which cell death is induced via various mechanisms in both p53-dependent and p53-independent manners