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28 For the specific classification, function, and mechanism of AATs in the human body, we invite readers to review the following literature

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Oxidative stress also activates the Kelch-like ECH-associated protein 1 (Keap1)-Nuclear factor erythroid 2-related factor 2 (Nrf2)-antioxidant response element (ARE) redox regulator pathway, releasing Nrf2 to regulate the expression of genes that control antioxidant, inflammatory and immune system responses, facilitating GSH activity

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Illustratively, BPC 157 therapy attenuated/eliminated the whole BuddChiari syndrome [26] (i.e., left superior caval vein-azygos vein-inferior caval vein shunt to skip the suprahepatic occlusion of the inferior caval vein), including the counteraction of the prominent deadly syndrome