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Hormonal alterations Since stress has been described as a potential trigger for ME/CFS (422) and cause of symptom flare-ups (423, 424), it is likely that the HPA axis- the neurobiological stress system- may be implicated in ME/CFS (53, 425, 426), as well as abnormalities in growth hormone (GH) secretion and dysfunctional adrenergic metabolism (255)

While low to moderate levels of ROS are essential in various physiological processes, their excessive and uncontrolled production causes failure of endogenous defences, leading to oxidative stress (OS) and possibly causing damage to the proteins, lipids, and DNA

Neuroprotection and neurodegenerative diseases: An emerging area of GHK-Cu research concerns its neuroprotective potential

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CD47 the surface protein CD47 is a receptor that binds to signal-regulatory protein alpha (SIRP) or thrombospondin-1 (TSP-1) and acts as a signal to inactivate the phagocytosis in macrophages

CC performed and analyzed the in silico experiments