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Upon binding to cytoplasmic glucocorticoid receptors (GR), corticosteroids induce translocation of GR complexes into the nucleus, where they interact with glucocorticoid response elements (GREs) to upregulate anti-inflammatory genes (e.g., IL-10, DUSP-1) and suppress pro-inflammatory mediators like IL-1, IL-6, TNF-, and chemokines, such as MCP-1 and IL-8 (McManus 2003

The three peptides together create what researchers call a synergistic healing cascade

MS raw data files were searched against the Uniprot human database (UP000005640, date:22/04/2020) using label-free quantitation algorithm of MaxQuant v1.6.5.0 44

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8A) and Akt phosphorylation at T308 (Fig