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Conversely, the release of cellular components or inflammatory signals following ferroptotic cell death can amplify immune responses, establishing a self-perpetuating immunometabolic crosstalk that may accelerate lesion progression and exacerbate tissue damage in OA

In accordance with our results, Saito et al

There is a C-to-T single-nucleotide polymorphism at the 718 position (rs713041) located in the 3-untranslated region of the GPX4 gene close to the predicted selenocysteine insertion sequence structure (SECIS, a region of the mRNA required for incorporation of the amino acid Sec into GPX4)

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European Journal of Clinical Investigation (Review)

Sterile, nonpyrogenic preparation for laboratory use